Limiting Salt Consumption – Can It Really Hurt You?

The study found that the limitation of salt consumption can even worse the consequences of a common heart failure.In addition to black and other ethnic minorities, young individuals seem to be at the most risk. Although the limitation of salt consumption is thought to be an important part of treating heart failure, research published in Heart magazine reveals that doing this very seriously can make things worse for those who have a common form of the situation.

The findings show that the most vulnerable groups are groups of young individuals, black and other ethnic origins.

Salt restriction is often recommended in heart failure guides, but the ideal range (less than 3 g per day) and is uncertain of the effect of patients with heart failure with preserved ejection fraction, because such patients are often left out.

With the preserved ejection fraction that forms half of the heart failure, the heart failure is the heart failure, the left ventricle, the lower left room of the heart, can not fill it properly with blood (diastolic phase), which causes less blood pumping into the body.

The researchers used the secondary analysis of 1713 Topcat trial participants, which are 50 years old or larger and To investigate the link between preserved ejection fraction and heart failure and heart failure due to heart failure salt consumption.

This study, which is a study of phase III, double blind, randomized, controlled work, is designed to learn whether the drug spironolactone can effectively treat symptomatic heart failure with preserved ejection fraction.

Participants were asked how much salt they added to cooking staples such as rice, pasta and potatoes; soup; meat; and vegetables and this: 0 points (no); 1 (⅛ teaspoon); 2 (¼ teaspoon); and 3 (½+teaspoon).

Their health was watched for 3 years for the primary last point. Routinely go to the hospital for a composition of death from cardiovascular disease or heart failure to maintain sudden heart stop. Secondary The consequences were admitted to the hospital for death and heart failure from any case.

Approximately half of the participants (816) cooking salt score was zero: more than half of them were male (56%) and most white ethnic origin (81%). They were significantly weighed more and had lower diastolic blood pressure (70 mm Hg) than those with a cooking salt score of over zero (897).

In addition, more often for heart failure, Type 2 diabetes, weaker kidney function to have, to control the heart sufficiency medication to control the left ventricular ejection fraction (lower cardiac output) was not less.

For the primary last point, the participants with a cooking salt score of over zero are significantly lower than the zero ones, and their probability of being adopted to a hospital for heart failure is lower. However, the possibility of dying from cardiovascular disease for any reason was not less than the zero with cooking salt score.

People aged 70 and under were significantly higher than adding salt to a hospital for accepting a hospital for primary end point and heart failure.

Similarly, those who came from black and other ethnic origins were small, although the numbers were small, they were more beneficial than adding salt to cooking compared to white ethnic origin.

Gender was not associated with the use of previous hospital admission and the use of heart failure drugs for heart failure, increasing risks of measured results and cooking salt score.

This is an observational study and therefore why cannot create. Not all data obtained from the Topcat experiment was not available, the cooking salt score was reported to itself and accepted the researchers. In addition, the reverse causality, which can be recommended to further restrict salt intake of people with lower health, cannot be ignored.

Low sodium intake is usually associated with low blood pressure and risk of cardiovascular disease in general public opinion and with high blood pressure. It is thought that it reduces fluid holding and triggering the hormones in the blood pressure regulation.

However, researchers say that the restriction of salt intake to control heart failure is less simple. It may cause intravascular volume contraction, which may reduce the need to alleviate the blockage and the need for liquid retention of water tablets.

However, the findings of the study show that the plasma volume in the blood is not significantly related to the scaling of the gauge-tanning salt score, which shows that low sodium intake does not alleviate fluid in people with heart failure with preserved ejection fraction, and pay attention to the researchers.